Guide S.0.S. SIMPLE OLD SOLUTION For Type 2 Diabetes

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In the patient group, the increase in the level of C-peptide after using honey was not significant when compared with glucose or sucrose. Significantly, the high degree of tolerance to honey was recorded in subjects with diabetes as well, indicating a lower glycemic index of honey. Honey elevated PGL after 1 hour and decreased it after 3 hours. Elevation of insulin and C-peptide was significantly higher after dextrose than after honey. Dextrose slightly reduced cholesterol and low-density lipoprotein cholesterol LDL-C after 1 hour and significantly after 2 hours and increased TG after 1, 2, and 3 hours.

Hypertriglyceridemic patients: artificial honey increased TG, but honey decreased TG. In diabetic patients, honey compared with dextrose caused a significantly lower rise of PGL. Honey caused elevation of insulin compared to sucrose after different intervals and lower elevation of PGL in diabetics. Longer-term honey consumption resulted also in weight reduction in all the patients, and control of the blood pressure in the patients, who had hypertension before the honey intervention. The cardiovascular status improved in the patients, who had coronary heart disease CHD before the intervention.

Both the GI and PII of honey were significantly lower when compared with sucrose in patients and controls. In both patients with diabetes and controls, the increase in the level of C-peptide after the honey was significant when compared with either glucose or sucrose. Honey in Diabetic Wound Healing Besides the health benefits of ingesting honey in diabetes, another important use of honey could be in managing diabetic wounds [ 89 ].

Conclusions Considerable evidence from experimental studies shows that the honey may provide benefits in the management of diabetes mellitus. Conflicts of Interest The authors declare that there are no conflicts of interest regarding the publication of this paper. Authors' Contributions All authors contributed equally to this paper.

References 1. Shaw J. Global estimates of the prevalence of diabetes for and Diabetic Research and Clinical Practice. Wei M. Effects of diabetes and level of glycemia on all-cause and cardiovascular mortality: the San Antonio Heart Study. Diabetes Care. Barr E. Risk of cardiovascular and all-cause mortality in individuals with diabetes mellitus, impaired fasting glucose and impaired glucose tolerance.

Kokil G. Pharmacology and chemistry of diabetes mellitus and antidiabetic drugs: a critical review.

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Mortality attributable to diabetes: estimates for the year Beretta G. Analytica Chimica Acta. Bertoncelj J.


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Evaluation of the phenolic content, antioxidant activity and color of Slovenian honey. Food Chemistry.

Only 2 Ingredients Say Goodbye to Diabetes Forever

Physico-chemical and bioactive properties of different floral origin honeys from Romania. Alvarez-Suarez J. Antioxidant and antimicrobial capacity of several monofloral Cuban honeys and their correlation with color, polyphenol content and other chemical compounds. Food and Chemical Toxicology. Erejuwa O. Antioxidant protective effect of glibenclamide and metformin in combination with honey in pancreas of streptozotocin induced diabetic rats. International Journal of Molecular Science. Hypoglycemic and antioxidant effects of honey supplementation in streptozotocin-induced diabetic rats.

International Journal for Vitamin and Nutrition Research. Hepatoprotective effect of Tualang honey supplementation in streptozotocin-induced diabetic rats. Galal R. Potential protective effect of honey against paracetamol-induced hepatotoxicity. Archives of Iranian Medicine.


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Asaduzzaman M. Effects of honey supplementation on hepatic and cardiovascular disease CVD marker in streptozotocin-induced diabetic rats. Rakha M. Cardioactive and vasoactive effects of natural wild honey against cardiac malperformance induced by hyperadrenergic activity. Journal of Medicinal Food. Yaghoobi N. Natural honey and cardiovascular risk factors; effects on blood glucose, colesterol, triacylglycerole, CRP and body weight compared with sucrose. The Scientific World Journal.

Khalil M. Cardioprotective effects of tualang honey: amelioration of cholesterol and cardiac enzymes levels. BioMed Research International. Weston R. The contribution of catalase and other natural products to the antibacterial activity of honey: a review. Tan H.

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The antibacterial properties of Malaysian Tualang honey against wound and enteric microorganisms in comparison to Manuka honey. Asian Pacific Journal of Tropical Biomedicine. Mohapatra D. Antibacterial efficacy of raw and processed honey. Biotechnology Research International. Hammond E. Antibacterial effect of Manuka honey on Clostridium difficile. BMC Research Notes. Nishio E. Antibacterial synergic effect of honey from two stingless bees: Scaptotrigona bipunctata Lepeletier, and S. Scientific Reports. An in vitro examination of the antioxidant and anti-inflammatory properties of buckwheat honey.

Journal of Wound Care. Kassim M. Ellagic acid, phenolic acids and flavonoids in Malaysian honey extracts demonstrate in vitro anti-inflammatory activity. Nutrition Research. Borsato D. Topical anti-inflammatory activity of a monofloral honey of Mimosa scabrella provided by Melipona marginata during winter in southern Brazil. Yaacob N. Tualang honey promotes apoptotic cell death induced by tamoxifen in breast cancer cell lines.

Evidence-Based Complementary and Alternatively Medicine. Honey as a source of dietary antioxidants: structures, bioavailability and evidence of protective effects against human chronic diseases. Bulut H.

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Curing Diabetes: How Type 2 Became an Accepted Lifestyle

Honey prevents oral mocositis in children undergoing chemotherapy: a quasi-experimental study with a control group. Complementary Therapies in Medicine. Porcza L. Honey and cancer: current status and future directions. Bahrami M. Effects of natural honey consumption in diabetic patients: an 8-week randomized clinical trial.

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Gastroparesis is a disorder affecting people with both type 1 and type 2 diabetes in which the stomach takes too long to empty its contents delayed gastric emptying. The vagus nerve controls the movement of food through the digestive tract. If the vagus nerve is damaged or stops working, the muscles of the stomach and intestines do not work normally, and the movement of food is slowed or stopped.

Just as with other types of neuropathy, diabetes can damage the vagus nerve if blood glucose levels remain high over a long period of time. High blood glucose causes chemical changes in nerves and damages the blood vessels that carry oxygen and nutrients to the nerves. Gastroparesis can make diabetes worse by making it more difficult to manage blood glucose.

When food that has been delayed in the stomach finally enters the small intestine and is absorbed, blood glucose levels rise. If food stays too long in the stomach, it can cause problems like bacterial overgrowth because the food has fermented. Also, the food can harden into solid masses called bezoars that may cause nausea, vomiting, and obstruction in the stomach. Bezoars can be dangerous if they block the passage of food into the small intestine.